The scientists found that alcohol affects the same chemical pathways in the brain fast acting as antidepressants, with the effects lasting 24 hours.
scientists warned that, despite the new findings, which are not suggesting alcohol can be considered as an effective treatment for depression.
But they have found to produce the same neural and molecular changes as drugs that have proven effective quickly.
Professor Kimberly Raab-Graham, of Wake Forest Baptist Medical Centre in the United States, said having a few drinks can help people with clinical depression feel better.
in animal experiments alcohol followed the same biochemical pathway as fast antidepressants, while producing effects similar to those seen in people behavior.
Prof Raab-Graham said: “Due to the high comorbidity between major depressive disorder and alcoholism is hypothesized widely recognized self-medication, suggesting depressed individuals may resort to drinking as a means. to treat their depression
“now we biochemical and behavioral to support this hypothesis data.”
however, said the findings, published in the journal Nature Communications ., does not suggest at all alcohol can be considered as an effective treatment for depression
Prof Raab-Graham said. ” definitely, there is a danger of self-medicate with alcohol
“There’s a fine line between what is useful and harmful, and at some point during repeated use of drugs self becomes an addiction.”
the researchers used an animal model to show a shot of alcohol, enough to block learning and memory, worked with other chemicals in the brain to trigger neural activity.
changes resulted in the prevention of depressive symptoms in animals that lasted for at least 24 hours.
in recent years, rapid single doses of antidepressants such as ketamine have proven to be able to .. relieving depressive symptoms within hours
this can last up to two weeks, even in individuals who are resistant to traditional antidepressants
Prof Raab-Graham added: “further research is needed in this area, but our results provide a biological basis for the natural instinct human being to self-medicate.
“also define a molecular mechanism that may be a critical factor for comorbidity that occurs with major depressive disorder and alcohol consumption.”